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IGF-1 LR3 interacts with growth signaling pathways involved in protein synthesis and cellular regeneration

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The compound enhances FAK autophosphorylation at Y397, creating high-affinity binding sites for Src family kinases

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The solution would be placed in appropriate dispenser for ingestion as a liquid or administration as a spray

In humans, IGF-I also increases renal blood flow and GFR by ~25% and causes volume expansion and sodium retention by a direct action on the renal tubules, with stimulation of rennin release and suppression of atrial natriuretic peptide secretion[364]