Various genetic polymorphisms in CYP enzymes and their levels of activity may explain why APAP is metabolized with excessive or diminished oxidative capabilities
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34 We demonstrated that tonic GABA currents, primarily driven by astrocytic GABA, 17 were significantly higher in PTSD-like mice compared to controls when measured following the administration of a GABAA receptor antagonist, bicuculline (50 M) (Fig
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