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A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington's disease in mice
In such a situation, oxidative stress may play an important role in the mechanism of neuronal death due to epilepsy
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For example, cyst-lining epithelial cells (the primary drivers of ADPKD progression) may exhibit distinct ferroptosis vulnerabilities compared to adjacent normal tubular epithelial cells, yet the molecular basis for this difference (e.g., unique lipid metabolic profiles and antioxidant capacity) has not been rigorously characterized
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