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This imbalance is strongly linked to the breakdown of the mucosal barrier and high systemic LPS levels (Wang L

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Glutathione-S-transferase (GSTP) that participates in the conjugation of GSH to reactive cysteines (S-glutathionylation) seems to play an important role in idiopathic pulmonary fibrosis lung fibrogenesis, since GSTP immunoreactivity is increased in the lungs of idiopathic pulmonary fibrosis patients, notably within type II epithelial cells (128, 129)