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Many people experience a slowdown in weight loss after 36 months of semaglutide therapy, a phenomenon called metabolic adaptation

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Oral semaglutide has led to more severe GI upset in all populations during trials [7, 18]

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GSH, the most abundant intracellular nonprotein thiol, plays a key role in intracellular defense, and decreased concentrations of this antioxidant are correlated with the increased frequency of reactive oxygen species (ROS)-mediated mitochondrial damage and apoptosis [9, 10]