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10.1016/j.bbrc.2015.11.129 67 ParkJ

Table 4 describes the overall incidence of immune-related adverse events by the type of checkpoint inhibitor

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The combination of GLP-1, GIP, and glucagon receptor agonism may further enhance these effects by collectively reducing the hepatic fat burden and systemic inflammatory state, thereby indirectly inhibiting the progression of fibrosis (47)

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