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The administration of flavonoids to diabetic animals boosts the antioxidant potential of -cells by increasing both enzymatic and non-enzymatic antioxidants, consequently limiting ROS generation and lipid peroxidation in -cells and protecting them against autophagy, apoptosis, or necroptosis (73, 74)

Clinically meaningful interactions would not be expected with other sulfonylureas (e.g., glipizide, tolbutamide, and glimepiride) which, like glyburide, are primarily eliminated by CYP2C9