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Consequently, this issue continues to be a focal point for clinical research, as understanding the exact uric acid levels that pose a risk is crucial for developing effective treatment and prevention strategies (5-7)

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Seiwerth and colleagues (Inflammopharmacology, 1999) documented effects on angiogenic growth factor pathway signalling, and subsequent work has expanded characterisation to include VEGF receptor 2 upregulation, endothelial cell migration in capillary-formation assays, and improved healing in ischemia-reperfusion preparations