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Because a major physiologic function of GSH is to provide cells with a reducing environment and to destroy the reactive oxygen compounds and free radicals formed in metabolism, organs that have low concentrations of other antioxidants (such as catalase and superoxide dismutase) are thought to be more dependent on GSH for detoxification of reactive oxygen species than are organs that have alternative antioxidants

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Identification of distinctive interferon gene signatures in different types of myositis

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In vitro research on cut tendons suggests that BPC-157 may enhance the migration of tendon fibroblasts to sites of injury [8]