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Recently the use of pharmacological agents which augment expression of Heme oxygenase 1 (HO-1) has been investigated

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It has to be stressed that chronic inflammatory conditions may be the basis for the insurgence of other pathological states, including cancer, broadening the role of OCTN2 and its main substrate, carnitine, in human pathology

Overcoming the compensatory increase in NRF2 induced by NPL4 inhibition enhances disulfiram/copper-induced oxidative stress and ferroptosis in renal cell carcinoma

Furthermore, it has been observed to counteract various forms of systemic damage and conditions, including tumor cachexia and the adverse effects of certain medical agents