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Osteoclast-derived exosomal miR-214-3p inhibits osteoblastic bone formation

Biological repair and adaptation rarely depend on a single signaling molecule, and many modern studies increasingly focus on network-level interactions rather than isolated mechanisms

Kwon HS, Koh SH

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In the hydrolysis mechanism (Figure 4), myrosinase cleaves the thioglycosidic bond, releasing -D-glucose and forming an unstable thiohydroxymate-O-sulfonate intermediate