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In hypertension, GAS5 is significantly downregulated in endothelial cells (ECs) and VSMCs, and its silencing exacerbates vascular remodeling, increases capillary leakage, and promotes endothelial dysfunction by modulating -catenin signaling

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I ironically was reading a book by Thomas Cowen, in which he explains in detail the purpose of fever and why it is important in our design to fight infection