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The nuclear translocation of damaged mitochondrial DNA activates the poly (ADP-ribose) polymerase 1 (PARP-1) pathway, resulting in inhibition of glyceraldehyde-3-phosphate dehydrogenase (GAPDH) activity, disruption of glycolysis[99], and marked upregulation of the p38 MAPK-cFOS inflammatory axis, which collectively promote endothelial inflammation[100]

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6ag and Supplementary Fig