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A study (52) has shown that baicalin can improve joint inflammation in CIA mice, inhibit the expansion of Th17 cells in the body, and down-regulate the expression of adhesion molecules (ICAM-1 and VCAM-1) and inflammatory factors (IL-6 and TNF-) in synovial cells stimulated by IL-17

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Despite advancements in standard-of-care therapiesincluding glycemic control, blood pressure management, and renin-angiotensin system blockadethe global burden of DKD continues to rise, underscoring the need for deeper mechanistic insight and novel therapeutic strategies